Hallmarks of AC disease pathogenesis and progression detected in human patients
Description
The central hallmark of AC is the fibro-fatty substitution of the myocardial tissue, being linked to the instability/disruption of intercalated discs (IDs) and desmosomes, the activation of FAPs (fibroblast activation proteins), the dysregulation of signalling pathways such as Wnt/β-catenin, and inflammation.
Acknowledgements
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